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DTSTART:19960101T000000 END:STANDARD BEGIN:STANDARD TZNAME:GMT TZOFFSETFROM:+0100 TZOFFSETTO:+0000 DTSTART:19961027T020000 RRULE:FREQ=YEARLY;BYMONTH=10;BYDAY=-1SU END:STANDARD END:VTIMEZONE BEGIN:VEVENT DTSTAMP:20260609T211756Z DTSTART;VALUE=DATE-TIME:20190410T150000 DTEND;VALUE=DATE-TIME:20190410T160000 SUMMARY:CDB Special Seminar: Mechanisms for rapid genome evolution\, Prof essor David Pellman\, TZID:Europe/London UID:20190410-8a1785d7699b61cd0169be7aeb7410a2@warwick.ac.uk CREATED:20190327T092840Z DESCRIPTION:David Pellman is the Margaret M. Dyson Professor of Pediatric Oncology at the Dana-Farber Cancer Institute\, a Professor of Cell Biol ogy at Harvard Medical School\, an Investigator of the Howard Hughes Med ical Institute\, and the Associate Director for Basic Science at the Dan a-Farber/Harvard Cancer Center. He received his undergraduate and medica l degrees from the University of Chicago. During medical school\, he tra ined with Dr. Hidesaburo Hanafusa at the Rockefeller University. His int ernship\, residency and fellowship in pediatric oncology were at Childre n’s Hospital and the Dana-Farber Cancer Institute. His postdoctoral fell owship was with Dr. Gerald Fink was at the Whitehead Institute and the M assachusetts Institute of Technology. Dr. Pellman has received the Damon Runyon Scholar Award\, the Stohlman Scholar Award from the Leukemia and Lymphoma Society of America\, the E. Mead Johnson Award and an NIH MERI T Award. He has been elected to the Association of American Physicians a nd is a fellow of the American Association for the Advancement of Scienc e. The laboratory has made contributions to understanding mechanisms of cell division and how cell division errors alter genome structure. Dr. P ellman’s accomplishments include: (1) the co-discovery of formin-depende nt actin assembly and a mechanism for positioning mitotic spindles withi n asymmetrically dividing cells\; (2) discoveries showing that whole gen ome duplication alters cell physiology\, can promote evolutionary adapta tion\, and can drive tumor development\; 3) the discovery of a mechanism explaining chromothripsis\, a recently discovered mutational process th at generates rapid karyotype evolution in cancer and congenital disease. LOCATION:GLT1\, Medical School Building CATEGORIES:BiomedicalSciences,BMS_CDB LAST-MODIFIED:20190327T092840Z ORGANIZER;CN=Jas Bains: END:VEVENT END:VCALENDAR