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BEGIN:VEVENT
DTSTAMP:20260508T084609Z
DTSTART;VALUE=DATE-TIME:20221207T131500
DTEND;VALUE=DATE-TIME:20221207T141500
SUMMARY:BMS Seminar: A simple and widespread mechanism for signal integra
 tion in bacteria? Professor David Grainger\, Professor of Molecular Micr
 obiology\, University of Birmingham
TZID:Europe/London
UID:20221207-8a17841b84c9b8ed0184d215b41b1e63@warwick.ac.uk
CREATED:20221202T090548Z
DESCRIPTION:Abstract: Vibrio cholerae is a Gram-negative bacterium respon
 sible for the human disease cholera. Estimates suggest 3 million annual 
 infections\, of which 100 thousand are fatal. Most disease instances are
  attributed to the El Tor V. cholerae biotype\, which is responsible for
  the ongoing 7th cholera pandemic. Globally\, over 1 billion people inha
 bit areas of endemicity and future climatic change is likely to exacerba
 te the risk of illness. The success of V. cholerae as a pathogen is unde
 rpinned by an ability to colonise both aquatic ecosystems and the human 
 intestinal tract. The transition between these environments will be the 
 focus of my talk. In particular\, I will discuss how global gene regulat
 ory networks can be integrated with each other via shared DNA binding si
 te specificity for transcription factors. I will discuss the possibility
  that this mechanism is a universal strategy utilised by many bacteria. 
 Biography: David started his lab in ÌÇÐÄTV in 2008 with the support of 
 a Wellcome Trust Career Development Fellowship. His group moved to Birmi
 ngham in 2011 where he is now Professor of Molecular Microbiology and a 
 Wellcome Investigator. He works on fundamental aspects of bacterial gene
  regulation\, antibiotic resistance mechanisms and control of pathogenic
 ity.
LOCATION:IBRB lecture theatre and via MS Teams
CATEGORIES:BiomedicalSciences,DivisionalSeminars
LAST-MODIFIED:20221202T090548Z
ORGANIZER;CN=Jas Bains:
END:VEVENT
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