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BEGIN:VEVENT
DTSTAMP:20260511T111056Z
DTSTART;VALUE=DATE-TIME:20200708T130000
DTEND;VALUE=DATE-TIME:20200708T133500
SUMMARY:Virtual BMS Divisional Seminar: Macrophages can either inhibit or
  enhance endometriosis depending on their origin\, Dr Erin Greaves\, Div
 ision of Biomedical Sciences\, ÌÇÐÄTV Medical School\, University of Wa
 rwick
TZID:Europe/London
UID:20200708-8a17841b71bb9cbc0171e58ddb5d7c02@warwick.ac.uk
CREATED:20200707T094523Z
DESCRIPTION:Abstract: Macrophages are intimately involved in the pathophy
 siology of endometriosis\, a chronic inflammatory disorder characterized
  by the growth of endometrial-like tissue (lesions) outside the uterus. 
 By combining genetic and pharmacological macrophage depletion strategies
  we determined the ontogeny and function of macrophages in a mouse model
  of induced endometriosis. We demonstrate that lesion-resident macrophag
 es are derived from eutopic endometrial tissue\, infiltrating large peri
 toneal macrophages (LpM) and monocytes. Endometriosis triggers continuou
 s recruitment of monocytes that seemingly contribute to LpM and small pe
 ritoneal macrophage (SpM) pools. Depletion of eutopic endometrial macrop
 hages results in smaller endometriosis lesions. Constitutive inhibition 
 of monocyte recruitment significantly reduces peritoneal macrophage popu
 lations and increases the number of lesions that develop. We propose a p
 utative model whereby endometrial macrophages and monocyte-derived macro
 phages\, possibly in LpM form\, are ‘anti-endometriosis’. These observat
 ions highlight the importance of monocyte-derived macrophages in limitin
 g disease progression. Zoom details can be found here:
LOCATION:Via Zoom
CATEGORIES:BiomedicalSciences,DivisionalSeminars,BMS_CDB
LAST-MODIFIED:20200707T094523Z
ORGANIZER;CN=Jas Bains:
END:VEVENT
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