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BEGIN:VEVENT
DTSTAMP:20260511T053731Z
DTSTART;VALUE=DATE-TIME:20201022T123000
DTEND;VALUE=DATE-TIME:20201022T133000
SUMMARY:BMS @ CSRL Seminar: Integration of Systems Biology with Tissue En
 gineering for Endometriosis Drug Development\, Professor Linda Gay Griff
 ith\, CMIT Center for Gynepathology Research
TZID:Europe/London
UID:20201022-8a17841a7520fdba01752c66ccd92cb3@warwick.ac.uk
CREATED:20201015T131536Z
DESCRIPTION:Abstract: ā€œMice are not little peopleā€ ā€“ a refrain becoming l
 ouder as the strengths and weaknesses of animal models of human disease 
 and drug responses become more apparent. At the same time\, three emergi
 ng approaches are headed toward integration: powerful systems biology an
 alysis of cell-cell and intracellular signaling networks in patient-deri
 ved samples\; 3D tissue engineered models of human organ systems\, often
  made from stem cells\; and micro-fluidic and meso-fluidic devices that 
 enable living systems to be sustained\, perturbed and analyzed for weeks
  in culture. Endometriosis\, adenomyosis\, and other gynecological disor
 ders are paradigms of chronic inflammatory diseases that can only partia
 lly be modeled in animal systems. In this talk\, approaches to classify 
 patients on the basis of analysis of cell-cell communication networks wi
 ll be described as a motivation for building patient avatars that captur
 e salient features of the disease processes. Then\, approaches to use sy
 nthetic extracellular matrices to build tissue-engineered models of the 
 endometrium and endometriosis lesions\, including microvascular networks
  and immune cell recruitment\, will be highlighted\, along with the pote
 ntial to integrate these approaches for developing new drugs to treat en
 dometriosis and adenomyosis.
LOCATION:Via MS Teams
CATEGORIES:BiomedicalSciences,DivisionalSeminars,CSRL
LAST-MODIFIED:20201015T131536Z
ORGANIZER;CN=Jas Bains:
END:VEVENT
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