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DTSTART:19960101T000000 END:STANDARD BEGIN:STANDARD TZNAME:GMT TZOFFSETFROM:+0100 TZOFFSETTO:+0000 DTSTART:19961027T020000 RRULE:FREQ=YEARLY;BYMONTH=10;BYDAY=-1SU END:STANDARD END:VTIMEZONE BEGIN:VEVENT DTSTAMP:20260615T232058Z DTSTART;VALUE=DATE-TIME:20160923T120000 DTEND;VALUE=DATE-TIME:20160923T130000 SUMMARY:Skin Microbiome Dependent Predisposition to Eczema TZID:Europe/London UID:20160923-094d434556d0030501570f0178b468bf@warwick.ac.uk CREATED:20160909T125217Z DESCRIPTION:Speaker: Dr Niranjan Nagarajan (National University of Singap ore) The human skin is our primary protection against environmental and pathogenic insults. Despite our best efforts to eradicate them\, our epi dermis harbours a complex microbial ecosystem. Different parts of the bo dy offer widely different environments\, the equivalent of variation fro m rain-forests to deserts for bacterial communities. While most skin res idents are harmless or beneficial to the host\, some can lead to skin di seases. In particular\, eczema of the skin is a common condition (15-30% of children and 2-10% of adults) where the interaction between skin mic robes and the host immune system is believed to play a role. Eczema or A topic Dermatitis (AD) has a significant quality of life impact on affect ed individuals and increasing healthcare and other associated costs worl dwide. Using modern genomic techniques and the first metagenome-wide ass ociation study on skin we have identified key perturbations in the resid ent skin microflora that could act as triggers for AD flares. This analy sis suggests that individuals with eczema harbour a distinct community t hat is primed for pathogen growth and inflammation. We propose a new for m of the hygiene hypothesis for explaining the increasing incidence of e czema\, revealing in the process novel therapeutic targets for this dise ase. LOCATION:MBU\, Medical School Building CATEGORIES: LAST-MODIFIED:20160909T132027Z ORGANIZER;CN=Becks Andrew: END:VEVENT END:VCALENDAR